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Addison’s Disease, A Simple Guide To The Condition, Treatment And Related Conditions
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- FormatePub
- ISBN978-1-370-04208-1
- EAN9781370042081
- Date de parution17/10/2016
- Protection num.pas de protection
- Infos supplémentairesepub
- ÉditeurEditeurs divers USA
Résumé
Addison Disease is a disease that is caused by the deficiency of circulating glucocorticosteroids and mineral corticoids (aldosterone) hormones as a result of disease of the adrenal glands. The causes of Addison Disease are:1. Auto-immune disease (Type 1 PGA - Poly-glandular Autoimmune Syndrome) of the adrenals with destruction of the adrenal parenchyma2. Tuberculosis of the adrenal glands3. Adrenal tumors that may be benign or malignant4.
Rare causes are hemorrhage, infarction, and infiltrative diseasesThe increased pigmentation especially in areas not exposed to the sun (such as buccal mucosa and palmar creases) and loss of weight can suggest the diagnosis of Addison DiseaseThis is due to excess pituitary production of ACTH which has melanocytes producing properties. There is also sparse growth of axillary and pubic hairs. Electrolytes especially sodium is low, potassium and urea nitrogen is highACTH stimulation test with impaired corticoid response is required to confirm diagnosisPlasma cortisol is low (< 3mcg/dL) and do not rise with ACTH stimulationAddison crisis is treated by intravenous normal saline and hydrocortisone 100mg every eight hoursTreatment must be done before laboratory confirmation of diagnosis because of the danger of comaChronic Addison disease requires replacement of glucocorticosteroids:Hydrocortisone 30mg daily (two thirds in the morning, one third in the evening)Cortisone acetate 37.5 mg daily orFludrocortisones 0.05 to 0.2 mg dailyDHEA 50mg orally in the morningAdequate salt intake is important for normal fluid and electrolyte balance.
Addison crisis patients have excellent recovery with appropriate treatmentChronic Addison disease patients also recovered well with replacement therapy. Care is needed when there is trauma, infection and surgeryTABLE OF CONTENTIntroductionChapter 1 Addison DiseaseChapter 2 Interesting Facts about Adrenal FatigueChapter 3 Treatment of Adrenal FatigueChapter 4 Adrenal FatigueChapter 5 Adrenal TumorsChapter 6 Adrenal CrisisChapter 7 Cushing SyndromeEpilogue
Rare causes are hemorrhage, infarction, and infiltrative diseasesThe increased pigmentation especially in areas not exposed to the sun (such as buccal mucosa and palmar creases) and loss of weight can suggest the diagnosis of Addison DiseaseThis is due to excess pituitary production of ACTH which has melanocytes producing properties. There is also sparse growth of axillary and pubic hairs. Electrolytes especially sodium is low, potassium and urea nitrogen is highACTH stimulation test with impaired corticoid response is required to confirm diagnosisPlasma cortisol is low (< 3mcg/dL) and do not rise with ACTH stimulationAddison crisis is treated by intravenous normal saline and hydrocortisone 100mg every eight hoursTreatment must be done before laboratory confirmation of diagnosis because of the danger of comaChronic Addison disease requires replacement of glucocorticosteroids:Hydrocortisone 30mg daily (two thirds in the morning, one third in the evening)Cortisone acetate 37.5 mg daily orFludrocortisones 0.05 to 0.2 mg dailyDHEA 50mg orally in the morningAdequate salt intake is important for normal fluid and electrolyte balance.
Addison crisis patients have excellent recovery with appropriate treatmentChronic Addison disease patients also recovered well with replacement therapy. Care is needed when there is trauma, infection and surgeryTABLE OF CONTENTIntroductionChapter 1 Addison DiseaseChapter 2 Interesting Facts about Adrenal FatigueChapter 3 Treatment of Adrenal FatigueChapter 4 Adrenal FatigueChapter 5 Adrenal TumorsChapter 6 Adrenal CrisisChapter 7 Cushing SyndromeEpilogue
A propos de Kenneth Kee
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