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The Social Void In Psychosis
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- FormatePub
- ISBN8235465015
- EAN9798235465015
- Date de parution17/05/2026
- Protection num.pas de protection
- Infos supplémentairesepub
- ÉditeurIoakim Ioakim
Résumé
The Social Void in Psychosis: Cognitive Mechanisms Underlying Loneliness in Schizophrenia Spectrum Disorders examines loneliness not as a secondary emotional consequence of social withdrawal, but as a primary, mechanistically driven feature of disrupted social cognition in schizophrenia spectrum conditions. The work integrates cognitive neuroscience, predictive processing theory, social cognition research, and phenomenological accounts of psychosis to propose a multi-level model of persistent subjective isolation.
Across schizophrenia spectrum disorders, loneliness emerges from the interaction of multiple dysfunctional systems rather than from simple lack of social contact. These include impaired predictive processing in social contexts, aberrant salience assignment mediated by dopaminergic dysregulation, disruptions in theory of mind and mentalization, instability in self-referential processing, and altered functioning within large-scale neural networks such as the default mode network, medial prefrontal cortex, and temporal-parietal junction.
Together, these alterations compromise the brain's ability to construct a stable, shared model of other minds. The resulting experience is characterized by a breakdown in social meaning-making, in which interpersonal cues become unpredictably weighted, emotional feedback becomes unreliable, and the distinction between internal and external sources of meaning becomes unstable. Social interaction may therefore be experienced as cognitively effortful, emotionally ambiguous, or perceptually distorted, contributing to progressive withdrawal and reinforcement of isolation.
Across schizophrenia spectrum disorders, loneliness emerges from the interaction of multiple dysfunctional systems rather than from simple lack of social contact. These include impaired predictive processing in social contexts, aberrant salience assignment mediated by dopaminergic dysregulation, disruptions in theory of mind and mentalization, instability in self-referential processing, and altered functioning within large-scale neural networks such as the default mode network, medial prefrontal cortex, and temporal-parietal junction.
Together, these alterations compromise the brain's ability to construct a stable, shared model of other minds. The resulting experience is characterized by a breakdown in social meaning-making, in which interpersonal cues become unpredictably weighted, emotional feedback becomes unreliable, and the distinction between internal and external sources of meaning becomes unstable. Social interaction may therefore be experienced as cognitively effortful, emotionally ambiguous, or perceptually distorted, contributing to progressive withdrawal and reinforcement of isolation.